Ustilago maydis
- 최초 등록일
- 2012.05.13
- 최종 저작일
- 2012.04
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MS 파워포인트
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소개글
깜부기병을 유발하는 Ustilago maydis의 유전자적 특징
목차
I. Introduction
II. Important tools for experimentation with Ustilago maydis
III. Cell fusion requires a complex signalling network
IV. Development of the dikaryon : the bE/bW complex at work
V. A connection between cell cycle, morphogenesis and virulence
VI. The early infection stages
VII. Proliferation and differentiation in the plant host
본문내용
VI. The early infection stages
V. A connection between cell cycle, morphogenesis and virulence
Appressorium Formation on the Plant Surface.
VI. The early infection stages
Gas1(alpha glucosidase)
Ten days after infection,
- Tumors are formed on plants infected with the wild type (left panel, arrow)
- Infection with Δgas1 strains leads to the formation of localized slightly necrotic
areas on the leaf (right panel, arrow).
VI. The early infection stages
Chs6, Chs7, and Mcs1 are essential during the infection process, In part, the pathogenicity of Δchs7 mutants could be restored when solopathogenic strains were used, indicating that the reduced virulence is partially attributable to the described mating defects. By contrast, Mcs1 activity becomes crucial when the fungus enters the plant.
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